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However, under oxidative stress conditions, Nrf2 dissociates from Keap1 and translocates to the nucleus, where it activates various antioxidant genes, including HO-1, protecting cells from oxidative damage

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In one study, TB-500 sulfoxide, produced by monocytes upon glucocorticoid administration, was found to inhibit neutrophil chemotaxis and exhibit potent anti-inflammatory effects [9]